Tuesday, January 16, 2018

HOW DO YOU SEE A LYME SPIROCHETE

WHAT THE MEDICAL WORLD SEES 



WHAT THOSE IN THE KNOW SEE 





Pharma only knows how to BLOCK mechanisms, not unblock them, but maybe something from chemotherapy to get rid of the mutated, incompetent B cells will work. Lyme is like a pseudolymphoma - incompetent B cells, immortalized (zombie B cells that wont die) from the EBV and also the shed fungal Osps/Vmps/etc.
This is important: People dont realized the essence of this. Fungal triacyl lipopeptides *ALSO* cause ***inhibition of apoptosis*** or appear to act like too much BCL2 class molecules, or like EBV-immortalized (or "transformed" is a word they use) lymphocytes. Spirochetes go right to the lymph nodes and cause "premature collapse of the B cell germinal centers (sort of a refinement school for new B cells formed in the bone marrow)" - which means they have not had antigen "presented" to them. Fungal lipopeptides do that - they cause antigen to not be presented by the "professional antigen-presenting cells," like macrophages and dendrocytes. They cause the downregulation of the HLA molecules (the clam shaped molecules that "present antigen" to the surface of the cell, against which antibodies are made).
This happens everywhere you run into this type of antigen (fungal, triacyl, TLR2/1-agonists), like Tuberculosis and mycoplasma. More on this mechanism is shown in the Occam's Razor and the Common Mechanisms charge sheets on truthcures.org.
Both spirochetes and EBV live (hang out, routinely) in the bone marrow, lymph nodes, and brain/nerves. I'd call that a clue as to how Lyme causes a chronic, AIDS like illness:
https://crymedisease.wordpress.com/…/inhibition-of-apoptos…/
Everything Allen Steere/Paul Duray describe here, is what we have been trying to explain to you is the disease: It is post-sepsis syndrome with reactivated EBV, mainly:
https://crymedisease.wordpress.com/…/steere-and-pseudolymp…/
"Reed-Sternberg cells" are basically EBV-transformed. And Steere and Duray even say, "this looks like mono (EBV)":
http://www.actionlyme.org/clinical-pathologic-correlations-…
The NIH has formally admitted that Chronic Lyme may be more about EBV in the New York Times. And THAT comes from the division who ran the "Lyme and Multiple Sclerosis" group. Another clue. And they found out this result was due to the shed fungal Osps. This too is in the charge sheets on truthcures.org and elsewhere in our blogs and websites.

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