Thursday, January 25, 2018

Cat's Claw

Cat's Claw

Herb; Cats Claw, uncaria tomentosa
This herb (inner bark) is an immune potentiator, anti-inflammatory, analgesic that has positive features for chronic Lyme infections that have become both immune disabling and brain central nervous system conditions.
It enhances cognitive brain functions and supports activity of the nervous system.
As immunity responses of chronic Lyme infection become impaired, certain features of blood antibody activity are noted to decline in specific categories.
Two important ones are CD57 and the natural killer NK cells.  Cats Claw helps to restore these cells for the body’s defense and this is very important.
Those who suffer neuroborelli’s brain infections will be aware of depressions.  Insomnia that frequently occurs and specific nerve symptoms ranging from slight tingles to grinding pain felt in the body in both left and right hemispheres at once in body zones.
The chronic brain fog and confusion that is sometimes experienced is very troubling for a psychological sense of well being.  Cat’s Claw can help with that problem.
It is specific to treat neurological pain that is often experienced with chronic Lyme disease.  It has a positive and supportive effect for the cognitive brain functions at the same time.
This being improved allows the mood or psychological condition to improve.  Insomnia will be less of a problem as stress declines and a normal sense of well being becomes possible again.
We do know how Lyme type bacteria manage to selectively manipulate the body’s immune system response. The spirochete sheds blebs of fungal antigens know as OSPA, OSPB etc. These fungal antigens suppress your immune system, basically turning it off. In turn any number of latent viruses may rear their ugly head. This is the reasoning behind everyone is different, true but not really. I fell we all deal with most of the same latent viruses, EBV, SIBO, Candida, Mycoplasma etc., etc., but at different strengths and times after the immune system lowers itself enough to be over whelmed by each one. Many experience multiple bacterial, viral attacks at once. From here we have a 50% chance of regaining control or a 50% chance of post sepsis, which at this point in time is irreversible. 
The ability of Cats Claw to help restore the NR and CD57 antibody immune cells means immunity will be brought back to a more normal condition of defense.
This will become a major road block to prevent progression of the disease.  No antibiotic can be more effective in fighting disease than natural immune capacity.
The history of medical use for Cats Claw is mostly involved with Native Americans and people who live within the South American geography, especially primitive jungle areas.  It can be rightly termed prehistoric but eventually explorers became aware of it and for us is a new discovery.
Cats Claw is becoming an important herb in post modern alternative medical use.  With Lyme type disease treatment it has a specific feature few other things offer.  Certainly not to this degree of effective immunity restorations for special antibodies.
The ability of Cats Claw essential.  Its remarkable positive effects on brain and nerve tissue should not be underestimated.  Yet immune system enablement may in fact be more important.
Apart from things previously mentioned, Cats Claw is a blood cleanser and system detoxifier and tonic.  It is hypo cholesterolemic and anticoagulant, antioxidant, antiviral and anti-tumor herb.  It shows promise for cancer treatment.
Usually used as a tincture. The tannins in the herb are released only if it is taken in an acidic medium; add a little lemon juice to a quarter-cup of water to which you add the tincture or prepare as a tea. For convenience it may be taken as a capsule.
NOTE:  This herb should not be used by pregnant women.  (Native women have used it to terminate unwanted pregnancy)  In large doses it can become an abortive herb.
The potential benefits of this herb should also be approached with a little caution.  This means go slow and build up dosage.
Above red highlighted paragraph has been rewritten by me. 

Wednesday, January 24, 2018

INTERESTING VIDEO ON LYME Anonymous - Biological Warfare (Lyme Disease)

Tuesday, January 23, 2018

LYMErix caused the same disease we know as “Chronic Lyme” Part 2

LYMErix caused the same disease we know as “Chronic Lyme” (Part 2)

….Continuing on to Part 2 
The LYMErix vaccine caused the same disease that us tick bite sepsis victims know as “Chronic Lyme disease”.  It was not taken off the market due to low sales.  The manufacturer SmithKline Beecham was given an ultimatum in 2002 by the FDA to take it off the market or they would.  This whole LYMErix fiasco is the reason that no one can get diagnosed or treated today.   I am shocked and astounded that anyone would even try to put out another vaccine for a disease caused by an organism capable of antigenic variation.  The French company Valneva is starting their phase 1 trials here in the United States and in Belgium this year.

OspA vaccination caused the same disease from a syringe as it does when you get Lyme disease from a tick bite.  LYMErix victims immune systems were destroyed by the vaccine because OspA is an endotoxin that causes immunosuppression and subsequent severe neurologic multi-system disease.
A wide range of neurological complications have been reported via the medical literature and the VAERS system after vaccination with recombinant outer surface protein A (OspA) of Borrelia. To explore this issue, 24 patients reporting neurological adverse events (AE) after vaccination with Lymerix, out of a group of 94 patients reporting adverse events after Lymerix vaccination, were examined for causation. Five reports of cerebral ischemia, two transient Ischemic attacks, five demyelinating events, two optic neuritis, two reports of transverse myelitis, and one non-specific demyelinating condition are evaluated in this paper.”
LUFT: “The point that I wanted to make in regard to the study is that there is very heavy dependence on serologic confirmation. “And when we start thinking about the adverse events, it was stated originally when we got the overview of the disease that the disease is really quite protean.  And actually the adverse events are very similar to what the disease manifestations are.”
  DR. PARENTI (SMITHKLINE):  Basically the two groups had the same suspect symptoms. We didn’t put it through statistical rigor, but when you looked at what it is that people came into the office with, what complaints, there was basically the same complaints in both groups.  So both groups were being evaluated for the same things.”
“Additional uncertainty may arise if the vaccines are not completely protective; vaccinated patients with multisystem complaints characteristic of later presentations of Lyme disease may be difficult to distinguish from patients with vaccine failure.”
OspA is Pam3Cys, a TLR 2/1 agonist which makes it an endotoxin.  What that means is that it causes permanent immune suppression.  This is referred to as “immune tolerance”, or “immune paralysis”, or when you read about the immune system becoming “overwhelmed”.  It creates a state of chronic sepsis, or post sepsis syndrome.  Once this happens viruses (that everyone has but can usually keep under control) like EBV and herpes reactivate and these are drivers of the “Great Imitator” outcomes we are all so familiar with.  Your body loses the ability to fight off other pathogens as well as the reactivation of latent viruses.  Injecting someone with an endotoxin like OspA and calling it a vaccine is criminal, plain and simple.  The reason the vaccine failed and caused these severe adverse reactions is because of what OspA is and does, being a TLR 2/1 agonist.
The similarity between the neurological sequelae observed in the OspA-vaccinated patients and those with chronic Lyme disease suggests a possible role for immune mechanisms in some of the manifestations of chronic Lyme disease that are resistant to antibiotic treatment.                                              
The criminals produced all of this data to market the vaccine saying how bad Lyme was to create a demand for their product.  We all needed to get this vaccine because the disease was so devastating and damaging to the brain.  The victims themselves said that this was the reason they got the vaccine in the first place, so it’s a little ironic that now it’s “anti-vaxxers” fault that people are contracting Lyme because there is no vaccine.
“I am unable to attend the January 31 FDA Vaccine Advisory Committee meeting due to a restrictive condition, Transverse Myelitis, resulting from the Lymerix vaccine. In the spring of 1999, I decided to get the series of Lymerix shots, after, viewing a very convincing TV commercial touting the importance of protecting oneself from Lyme Disease. I felt this would be a good thing to take advantage of since I had had numerous bites from the ticks which cause Lyme Disease.”
The reason that this fraud was committed was solely to qualify the LYMErix vaccine, they knew as early as the phase 1 trials that OspA was causing the same disease.  The plan the entire time was to make Lyme disease only 15% detectable, and to do that they needed to change the testing and the case definition.  If they said that the 85% of immunosuppression neurologic outcomes didn’t exist then they could say that they vaccine was 85% effective.  It’s really that simple.  They realized that there were two types of the disease, so they cut one of them out effectively ruining millions of lives.  They saw an opportunity to corner the market by patenting all of the different tick borne diseases and only allow certain labs to test the blood.  (They wanted all of the blood to go only to them so they could find other diseases to patent for vaccines and test kits, but thats another story for another post.)
Before this master plan was put into motion Allen Steere wrote all about deformed B cells with Paul Duray.  Lyme Borreliosis went from being a severe multi-system neurologic disease largely driven by immune suppression/dysfunction to being Lyme disease with just an arthritic knee and robust antibody production.
The plasma cell precursors are large, appear tumor-like, and can resemble Reed-Sternberg cellsOthers look like typical immunoblasts (FIG. 1). In one example, cervical lymph nodes show cell degeneration with karyorrhexis and nuclear debris of lymphoid elements. This patient had repeated high fevers and marked discomfort of neck nodes. Large atypical immunoblasts can also be seen in the spleen and bone marrow. The red pulp of the spleen is congested, not unlike that seen in infectious mononucleosis. Spirochetes can be demonstrated in the lymph nodes, spleen and bone marrow and liver.”
That certainly doesn’t sound like an easy to cure illness with 21 days of doxy to me..
”…when lymphocytes are cultured in the presence of growing Bb there is a marked inhibition ( p < . 0005 ) of NK activity on days 3, 5, and 7 when compared to lymphocytes cultured in BSKII media in the absence of spirochetes. This effect is not due to a selective depletion or toxicity to endogenous NK since viability studies and monoclonal antibodies demonstrate no significant changes after culture with the organism.  The inhibition is directly attributable to the organism or its supernatants [lipid layer] (data not shown).” 
“OspA interferes with the response of lymphocytes to proliferative stimuli including a blocking of cell cycle phase progression.”
Immune suppression.  Dattwyler says that the lower the antibody production (seronegativity) the sicker the patient is.
Dr.O’BRIEN: “I was concerned about your last slide where you said there was a poor correlation between serologic response and clinical disease. And as I heard you to say, some people who mount better immune responses get worse disease. Did I hear you say that?”
DR. DATTWYLER: “No, no, I said the reverse. The better responses tended to have better response. And I should clarify where this is from. This is from antibiotic trials. These are treatment trials of erythema migrans, in which individuals given an antibiotic regimen which was not optimal – we did not know that it was not optimal at the time – the ones that failed to mount a vigorous response tended to do worse, clinically. So, there was an inverse correlation between the degree of serologic response and the outcome.”                              So, individuals with a poor immune response tend to have worse disease.

There you have it.  Everyone is saying that tick bite Lyme disease and LYMErix cause immune suppression and severe debilitating illness.  This is just a tiny snapshot of the data compared to what is in the Charge Sheets on TruthCures website.  As always, I encourage you all to go there and check them out in their entirety.

Sunday, January 21, 2018

You Cannot Have a Lyme Vaccine.Part 1

You Cannot Have a Lyme Vaccine.

In light of the FDA’s recent fast track for the new Lyme disease vaccine brought to us by Valneva, which is still made out of OspA, we need to to talk about this.
So what are spirochetes?  How do they work, what do they do?  Here are some pictures to help us:

Do you see all the little bubble things?  Those are the blebs, and spirochetes shed them and this is what antigenic variation is.  Having this ability allows them to evade the hosts immune system pretty dang effectively as most of us already know.  Spirochetes are exceptional self preservationists.
“Many researchers believe that the secret to B. burgdorferi’s infectivity and inflammatory capacity lies in the interaction of its surface proteins with the host’s immunological system. Yale researcher Stephen Barthold, a veterinarian and professor of comparative medicine who developed the first mouse model of Lyme disease, studies the expression of B. burgdorferi surface proteins throughout various stages of the spirochete’s life cycle. He finds that during the early stages of infection, B. burgdorferi avoids immune detection by decreasing its expression of surface proteins or cloaking its expressed surface proteins under a layer of slime. “It’s using some sort of stealth-bomber-type mechanism,” he says. Or, using another diversionary tactic called blebbing, the spirochete can pinch off bits of its membrane in order to release its surface proteins. Explains Barbour: “It’s like a bacterial Star Wars defense program,” in which released surface proteins might intercept incoming host antibodies, keeping the spirochete safe from immunological attack.”
As you can imagine this antigenic variation stuff keeps the host immune system pretty busy.  The hosts immune system has to constantly adapt to the different outer surface proteins (Osps) yet having one antibody like OspA, say the makers of LYMErix, doesn’t actually do any good if spirochetes get inside of the body.  They say that it works by something so asinine it’s a quandary how they even got this sh*t on the market in the first place.
“These and other findings indicate that the B. burgdorferi spirochetes alter antigen expressionduring infection so as to evade the antibody response and do not elicit effective memory responses to protective antigens (i.e., those that are expressed by all spirochetes and likely essential for infectivity). Thus, identification of suitable antigens for induction of protective immunity has been a challenge.”
They know that spirochetes do this antigenic variation thing, and that its why antibodies to just one Osp are useless inside the human body.  They say that LYMErix works by disinfecting the spirochetes inside of the tick gut.  The tick attaches, starts drinking the hosts blood, the OspA antibodies get inside the tick and kill all of the spirochetes before the tick regurgitates them back into the host.
“A Lyme vaccine based on OspA, which was recently approved for human use, is an example of the former. When a tick feeds on a OspA-vaccinated individual or animal, antibodies that have developed in response to the vaccine enter the tick gut and kill the bacteria inside the gut before the population has a chance to diversify. This prevents the transmission process from taking place, de Silva explained.”
(It’s not as slow as they say it is for transmission,  but thats for an upcoming post)

What they fail to mention is that OspA is Pam3Cys.  Because if they did then they’d have to admit that they committed one of the most heinous medical crymes in human history and waltz into prison willingly charge with crimes that carry life sentences.
OspA = Pam3Cys = immunosuppression and immune tolerance = reactivated viruses and opportunistics = multiple severe neurolgic diseases occurring simultaneously = life ruined.  
OspA is a triacylated lipoprotein, which makes it a very toxic endotoxin (more so than LPS) and a TLR 2/1 agonist.  I’m sure you’re probably thinking “….what the heck is that and why should I care?”.  A triacylated lipoprotein is very toxic to humans, its an endotoxin.  Everyone says so, it’s common knowledge.  A TLR 2/1 agonist is also very bad.  To call something like this a vaccine is completely erroneous and just plain stupid not to mention dangerous.  LYMErix injured it’s victims pretty severely, it gave them the same disease we know as Chronic Lyme.pam3cys image
Just like the graphic says these shed blebs, being an endotoxin, turns off the immune system because if it didn’t you would die.  Voila!  Just like that you have something awful called Post Sepsis Syndrome!  More commonly known as Chronic Lyme disease, ME/CFS, Fibromyalgia, GWI and all the other great imitator outcomes.  Once the immune system down regulates, or shuts off, or is paralyzed, or is overwhelmed, or develops tolerance, or whatever you want to call this state of chronic sepsis- all of the latent viruses that 90% of the population have reactivate and start causing problems.  Like EBV, CMV, all of the Herpes.  EBV is known as the cancer virus and Herpes are found in the brain tissue of people who died from diseases like Multiple Sclerosis.
“Herpes simplex virus was present in more MS cases than control cases and in more active plaques than inactive plaques.”
The Role of Epstein Barr Virus in Cancer.
So this is no secret, it’s common knowledge.  And it’s criminal.  Need I remind everyone that RICO charges carry life sentences?  These idiots picked the dumbest thing on the planet to try to pass off as a vaccine.  The LYMErix victims never got compensation and were slandered and libeled every step of the way.  They are still putting nonsense out there that the vaccine was removed due to “low sales”, but we know the truth and TruthCures has the data to prove it.  All of this victim blaming makes it even more crystal clear that all of these criminals are COWARDS.
“I recently wrote about the rise and fall of LYMErix™ (also summarized here, the only medical product ever withdrawn from the market not because of science but because of the incredible public to-do about it. The intense controversy may seem odd, especially since we have had a trustworthy and no-big-deal Lyme vaccine for dogs for awhile now; but the leap from man’s best friend to man proved complicated. The vaccine somehow was forced to swim against the riptide caused by both the pro-chronic Lyme crowd who regard the vaccine to be as bad as the disease,and the equally forceful anti-any-vaccine contingent.”
Okay.  So Lyme isn’t a serious disease and is easily cured by antibiotics, so why do we even need a vaccine?  OH YEAH.  BECAUSE $$$$$
Here all the victims stories in their own words: in Brain tissueThis is a graphic of spirochetes in brain tissue, I have a whole post on spirochetes effects on brains.  Lyme disease is a permanent brain infection, but then it’s just a bad knee and people are catching hypochondria only from ticks, but get the vaccine because it’s a very serious disease.  How can they even keep up with their own bullsh*t?  You know what they say, once you start lying you have to keep lying so as not to show your lapse in integrity..

Thursday, January 18, 2018

The prevalence and significance of Borrelia Burgdorferi in the urine of feral reservoir hosts

The prevalence and significance of Borrelia burgdorferi in the urine of feral reservoir hosts.


Live Borrelia burgdorferi were isolated from the blood and/or urine of white-footed mice (Peromyscus leucopus) collected on Shelter Island, New York, in 1984 and 1985. Prevalence of spirochetes in urine was consistently higher than in blood or both fluids simultaneously. Spirochetes remained viable for 18-24 hours in urine and were maintained in culture for one week. Mice removed from the field were spirocheturic for at least 13 months. One spirocheturic mouse developed spirochetemia one month after field removal indicating the pathogen can return to the peripheral circulation. Twenty-one kidneys from 22 mice had spirochetes in the interstitial areas and bridging the tubules. A positive correlation between Babesia microti infection and spirocheturia was seen. Although the mechanism of entry into the urine is unknown, B. microti infection may increase glomerular permeability. Babesia induced hematuria may provide possible nutrients to maintain spirochetes. Urine may provide a method for contact non-tick transmission of B. burgdorferi in natural rodent populations particularly during periods of nesting and/or breeding.
[Indexed for MEDLINE]

Tuesday, January 16, 2018




Pharma only knows how to BLOCK mechanisms, not unblock them, but maybe something from chemotherapy to get rid of the mutated, incompetent B cells will work. Lyme is like a pseudolymphoma - incompetent B cells, immortalized (zombie B cells that wont die) from the EBV and also the shed fungal Osps/Vmps/etc.
This is important: People dont realized the essence of this. Fungal triacyl lipopeptides *ALSO* cause ***inhibition of apoptosis*** or appear to act like too much BCL2 class molecules, or like EBV-immortalized (or "transformed" is a word they use) lymphocytes. Spirochetes go right to the lymph nodes and cause "premature collapse of the B cell germinal centers (sort of a refinement school for new B cells formed in the bone marrow)" - which means they have not had antigen "presented" to them. Fungal lipopeptides do that - they cause antigen to not be presented by the "professional antigen-presenting cells," like macrophages and dendrocytes. They cause the downregulation of the HLA molecules (the clam shaped molecules that "present antigen" to the surface of the cell, against which antibodies are made).
This happens everywhere you run into this type of antigen (fungal, triacyl, TLR2/1-agonists), like Tuberculosis and mycoplasma. More on this mechanism is shown in the Occam's Razor and the Common Mechanisms charge sheets on
Both spirochetes and EBV live (hang out, routinely) in the bone marrow, lymph nodes, and brain/nerves. I'd call that a clue as to how Lyme causes a chronic, AIDS like illness:…/inhibition-of-apoptos…/
Everything Allen Steere/Paul Duray describe here, is what we have been trying to explain to you is the disease: It is post-sepsis syndrome with reactivated EBV, mainly:…/steere-and-pseudolymp…/
"Reed-Sternberg cells" are basically EBV-transformed. And Steere and Duray even say, "this looks like mono (EBV)":…
The NIH has formally admitted that Chronic Lyme may be more about EBV in the New York Times. And THAT comes from the division who ran the "Lyme and Multiple Sclerosis" group. Another clue. And they found out this result was due to the shed fungal Osps. This too is in the charge sheets on and elsewhere in our blogs and websites.

Thursday, January 11, 2018

Aluminum Adjuvant Causes Brain Inflammation and Behavioral Abnormalities; Low Dose Is More Harmful

A new paper (Crepeaux et al.) by the Gherardi research group in France reports important results on the toxicity and transport of aluminum (Al) adjuvant in mice. This study is especially valuable because it looked at many outcomes: behavioral effects, immune (microglial) activation in the brain, and Al transport into the brain. The study tested dosages of 200 , 400 and 800 mcg/Kg (mcg=micrograms, mcg/Kg = micrograms per kilogram of animal body weight),  injected intramuscularly (IM). The Al adjuvant used was AlOH (brand name Alhydrogel), the most commonly used vaccine adjuvant. It is in the tetanus, Hep A, Hep B, HiB, pneumococcal, meningococcal, and anthrax vaccines.
Remarkably, the study  found that the lowest dosage (200 mcg/Kg) was the most toxic! The 400 and 800 mcg/Kg dosages produced no statistically significant effects, but the 200 mcg/Kg dosage did.

Wednesday, January 10, 2018


    I wish it all a nightmare to awaken from, but it is all to real.  As I lose our home this battle with Lyme seems to have beaten me up at every turn. The victories come with a whole set of new challenges, many physical, many mental and frankly much that is simply too bazaar to try and explain.

  Disability that I desperately sought and received, first time on my own was a huge blessing at the time. Thank God for relief of suddenly overcoming the immense anxiety of being flat broke. The realization of absolutely no money is simply impossible to comprehend unless you experience it. Exactly much like Lyme, it is impossible to comprehend unless you live it. Yet once you acquire the disability insurance it the joy dissipates to the feelings of uselessness to one your family and two to society. My handicapped sticker is not a badge of honor I display proudly. Quite embarrassing if I were to be quite honest. The realization of being truly disabled took me some time. It's denial at first, then it is the slow agony of learning you are truly disabled. the smallest of tasks completed is akin to climbing a mountain. The pain and agony of letting your wife and daughter down is unbearable. I am so truly sorry for this.

   Family has become our greatest adversary. The plain cruelty is extremely harmful to my wife and daughter. For me it comes as no surprise. I can only surmise that the devil has surely taken over their being. You would think they have no other life but evil. But reflecting a bit more, a few have no life to speak of. Idle minds is the devils workshop, this certainly holds true in our case.

    While my most horrendous symptoms are settled down, my body and mind are left lacking. Much of which has come from western medicine. In this so called world of modern medicine anyone suffering from chronic pain is declared depressed. SSRI's are given for pain. Surely it is all in your head, a chemical imbalance they say. But I'm hear because of a botched knee replacement, severe pain in my knee and foot. So I'm given an anti depressant, how can I ever thank you doc. Maybe I will try putting them in my sock. Next visit the old PA switcharoo now I get another antidepressant and my range of motion in my foot is due to lack of effort. Nothing like a pompous a__ wanna be. I can't count the number of times I've played this game.

   I'm writing this to remind you that we are all in this cruel reality together. We are simply disposable now that we are not producing for the rest of the world. Yet I believe by joining all the victims of modern medicine together under one umbrella we can change this corrupt abusive medical system many of us face.   There is one thing for certain is that they'll be where we are one day.

                                         You ready to rise up yet?

Monday, January 8, 2018

Lyme Crymeline of the Worst Human Rights Abuses in History

Tuesday, November 7, 2017

Lyme Crymeline of the Worst Human Rights Abuses in History

How much longer are we going to keep running from the truth? When will we rise up to this Medical Holocaust? When is enough enough? How many more lives must be stolen? Childhoods destroyed and robbed? How many more warriors must die? Starve? Go homeless? Commit suicide? Medically kidnapped?  The atrocious crimes, genocide and murder committed by CDC officers for profit and greed have been layed out on a silver platter by the whistleblower herself for us all. Lymeland, it's time for us all to rise up, we are in the fight of our life. ILADS and non-profits won't stand up for us, they continue running away from the truth. We're alone in this fight, it's up to us and only us. Below is a breakdown of the Lyme Crymes to humanity generated by TruthCures

1980: The Bayh-Dole Act is passed.

This event set the stage for the gold rush to the patent office--not only in the U.S., but also in Europe, as evidenced by Barbara Johnson's (CDC officer) multiple patents with SmithKline in Europe. We also saw Alan Barbour's 30+ patents, and Yale's patents for both LYMErix (5,747,294) and the only valid test method for diagnosing both bad-knee Lyme and neuroborreliosis (5,618,533).
It became clear at this time that the mission of the Centers for Disease Control and Prevention became laser-focused on "prevention" by way of vaccines. 
Here is a paper by MIT student David Levenson on the consequences of the Bayh-Dole Act. 

1986: Allen Steere forms the basis for the 1990 case definition.

Here, in 1986, Steere not only says you only need band 41 for diagnosis, but he says treatment fails in half the cases. 
 This was the source of the first, 1990, CDC surveillance criteria - just do repeat Western Blots to look for new IgM bands, because that meant the bug was still alive after treatment.
"In contrast, among six patients with prolonged illness, the IgM response to the 41-kD protein sometimes persisted for months to years, and late in the illness during arthritis, a new IgM response sometimes developed to a 34-kD component of the organism. The IgG response in these patients appeared in a characteristic sequential pattern over months to years to as many as 11 spirochetes antigens. The appearance of a new IgM response and the expansion of the IgG response late in the illness, and the lack of such responses in patients with early disease alone, suggest that B. burgdorferi remains alive throughout the illness."

1986: Alan Barbour publishes “THE BIOLOGY OF BORRELIA SPECIES”
In this report, Alan Barbour, CDC officer, states:
"The propensity for borrelia to go to the brain of infected mammals suggests that the relationship between these spirochetes and neural tissues is not trivial.  Further study of this attraction and the interaction that follows may reveal the basis for the significant nerve and brain involvement in Lyme borreliosis."
All the way back in 1986 it is not trivial that Borrelia have an affinity for brains.  It is also not trivial that you cannot kill spirochetes, and that the nature of the disease as a Relapsing Fever germ is in fact, to relapse. This paper also states that it was attempted to treat syphilis with a wimpy high passage strain of Borrelia to cause fever because antibiotics didn’t work.
Barbour later became the patent king of tick-borne diseases. 

1986: Allen Steere talking about how Lyme is like post-sepsis with the leukemia-like, EBV-transformed immune cells.

 In this report, Allen Steere states:
"Not only are plasma cells plentiful in the spleen, lymph nodes and bone marrow, they are also represented by large and somewhat atypical-appearing precursor B cells as well."
"Numerous names have been given to this stage, including pseudolymphoma, lymphoid hyperplasia, follicular hyperplasia, lymphocy- toma cutis, Spiegler-Fendt lymphoid hyperplasia, and lymphadenosis benigna cutis of Baverstedt."
"The immune response involves virtually all of the organs and structures of the reticuloendothelial system including the bone marrow, and clinical pain and discomfort seems to correlate with hyperplasia of lymph nodes and spleen and bone marrow. Diffuse visceral involvement in this acute stage mimics infectious mononucleosis or disseminated viral syndromes. These include conjuctivitis, pharyngitis, pneumonitis with dry cough and mild pleuritic pain, hepato-splenic tenderness, lymph node swelling of the neck and groin, and orchitis. There is lymphoid hyperplasia of the lymph nodes and spleen consisting of prominent germinal centers and numerous perifollicular lymphocytes, with prolifera- tion of plasma cell precursors and mature plasma cells. The plasma cell precursors are large, appear tumor-like, and can resemble Reed-Sternberg cells."
Lyme is like cancer, he says. 

1986: Edward McSweegan's fake whistleblower letter to steal tick-borne disease funding from the Navy.

 Here Edward McSweegan writes a fake whistleblower letter intending to steal the US Navy's (his employer) funding and give it to all his cronies at the future American Lyme Disease Foundation (ALDF).  
 He does not know spirochetes undergo antigenic variation or that its surface antigens, formerly called "mucopeptides," were fungal-type antigens and therefore a vaccine can't be made out of them.  It's the same for most of the other tick-borne diseases; they are bearers of fungal antigens, which is why they were always known to make nice bioweapons.

1988: Raymond Dattwyler associates immunosuppression with borrelial lipoproteins.
 In this report, Dattwyler states:
"Figure 1 demonstrates that there was a marked and highly significant inhibition ( p < 0.0005) of invivo endogeneous NK activity in two patient populations: (1) patients in the early stages of Lyme disease (ECM stage) and before treatment, and (2) patients exhibiting chronic active stages of the disease."
Natural Killer Cell activity is muted, and Dattwyler also says Borrelial supernatant (where the fatty Osps are) does this too.  That means it was known in 1988 that Borrelia and its shed mucopeptides or lipoproteins cause immune system blunting.

1988: Raymond Dattwyler develops a seronegative Lyme assay.
 In this report, Dattwyler states:
“We studied 17 patients who had presented with acute Lyme disease and received prompt treatment with oral antibiotics, but in whom chronic Lyme disease subsequently developed. Although these patients had clinically active disease, none had diagnostic levels of antibodies to B. burgdorferi on either a standard enzyme-linked immunosorbent assay or immunofluorescence assay. On Western blot analysis, the level of immunoglobulin reactivity against B. burgdorferi in serum from these patients was no greater than that in serum from normal controls.”
Here, knowing Lyme (especially chronic neurologic Lyme) is seronegative, they develop an assay that Steere later uses to assess "Chronic Neurologic Lyme" cases, proving he knows these cases are seronegative.

1989: Duray publishes in IDSA's journal that chronic Lyme is like syphilis and may be permanent.
 In this report, Duray states:
“Nervous system involvement. Biopsy of the sural nerve in patients withe peripheral neuropathy associated with chronic Lyme disease has demonstrated the presence of lymphocytes and plasma cells in the nerve itself as well as in the perineurial region (figure 10). This is not dissimilar to the pattern of infiltrates seen in stage II. What differentiates this complication from stage II involvement, however, is its apparent permanence.”
“Further complicating the staging of this infection is the fact that serologic tests often do not show any correlation between titer and extent of damage to organ systems.”

 1990: The ALDF is formed.
 The American Lyme Disease Foundation (ALDF) was formed apparently for the purposes of spinning Lyme disease as merely a nuisance disease that at the same time, was serious enough that we needed a vaccine to prevent it. It is the racketeering organization at the center of the Lyme Cryme. 
In an article from New York Medical College, where the ALDF was conceived, Arthur Weinstein explains its founding:
"This year, Dr. Weinstein is working with more than half a million dollars in grants from the NIH, pharmaceutical companies and private foundations on research that includes systemic lupus, rheumatoid arthritis and scleroderma. But the bulk of the funds belong to chronic Lyme disease, which was not his interest when he joined New York Medical College from the University of Connecticut School of Medicine in 1985; lupus was. Still, Dr. Weinstein became intimately involved in the evolution of Lyme here as his experience with the disease went from nothing to plenty, and fast.
"The College has recognized that Lyme disease is a major clinical and research interest on this campus. The main players in the development of the program were Fish, Wormser and Connolly," Dr. Weinstein advises. The entrepreneurial trio are Durland Fish, Ph.D., former director of the College's Lyme Disease Center and now a research scientist at Yale; Gary P. Wormser, M.D., still professor of medicine and pharmacology and chief of the Division of Infectious Diseases at the College; and John J. Connolly, Ed.D., former College president and current chairman of the board of the American Lyme Disease Foundation, Inc., which had its genesis on the Valhalla campus in 1990."
Click here to see a 990 tax form that shows the ALDF receiving grant money from the CDC.

1990: Steere uses the Dattwyler Seronegative Lyme Assay to assess Chronic (Seronegative) Post-Tick Bite Sepsis or Neurological Lyme.

 In this report, Allen Steere states:
“Of the 27 patients, 24 (89%) had a mild encephalopathy that began 1 month to 14 years after the onset of the disease and was characterized by memory loss, mood changes, or sleep disturbance. Of the 24 patients, 14 had memory impairment on neuropsychological tests, and 18 had increased cerebrospinal fluid protein levels, evidence of intrathecal production of antibody to B. burgdorferi, or both. Nineteen of the 27 patients (70%) had polyneuropathy with radicular pain or distal paresthesias; all but two of these patients also had encephalopathy. In 16 patients electrophysiologic testing showed an axonal polyneuropathy…Among the 27 patients, associated symptoms included fatigue (74 percent), headache (48 percent), arthritis (37 percent), and hearing loss (15 percent).”

1990: CDC publishes a Lyme case definition that recognizes it as "relapsing fever."

 Here the published surveillance case definition is based on Allen Steere's 1986 report, "Antigens of Borrella burgdorferi Recognized during Lyme Disease."
 They say, just do repeat Western Blots to look for new IgM bands, because that meant the bug was still alive after treatment.
 See Steere's page here
To be clear, all Borreliae are relapsing fever organisms, and the nature of the relapse is antigenic variation. Therefore you cannot use any DNA from plasmids – which is where the variable surface antigens are ordered manufactured and remanufactured – to assess for spirochetes. No one sane does this. No researchers outside the United States EVER uses plasmid DNA to assess for spirochetes. They only use species-specific spacer genes like 5, 16 and 23 S RNA or flagellin. That’s it.

1991: Yale's Fikrig & Flavell patent the only valid test method for all "versions" of "Lyme disease."

The current (since 1994) testing method for “Lyme disease” does not make sense when you take into account that Borrelia are relapsing fever germs.  Borrelia are differentiated by their Flagellin genes (DNA).  Since all Relapsing Fever organisms are capable of antigenic variation (they can change their outer surface proteins  as a way of evading the host’s immune system) the only scientifically VALID way to test for Borrelia infections would be to test for Flagellar antibodies instead of outer surface proteins, because it’s specific and never changes.
Testing for so many antibodies all at the same time is NOT logical, or valid, by any standard.  
This patented test is 94%-95% accurate during all stages of the disease.  This is the test that should be used, obviously.  
Here is the report from Yale that goes with the patent, U.S. Patent 5,618.533.

1991: Allen Steere says he is "convinced seronegative Lyme exists."

... and that this non-HLA linked outcome is "perilously close to Fibromyalgia and Chronic Fatigue Syndrome." In this report, Allen Steere states:
"Most of these patients have subtle encephalopathy affecting the central nervous system. They have memory difficulty, depression, or sleep disturbances but no seizures, myoclonus, or changes in the level of consciousness. They also have sensory symptoms, such as pain in the spine, accompanied by radicular pain in the limbs or trunk, and some have distal parethesias with intermittant tingling sensations in the hands and feet...These symptoms are perilously close to those that occur in fibromyalgia, with the chronic fatigue syndrome, or in stress-induced syndromes-conditions that are ever so much more common than tertiary Lyme disease.
 "How then does one identify the patient with chronic neurologic abnormalities of Lyme disease?
 "The patients in question have characteristic findings on laboratory evaluations as follows:almost all were sero-positive by ELISA (keep in mind, this is PRIOR TO his research fraud in Europe in which he falsified the ELISA and added it as a bogus screening test.), half of them had increased cerebrospinal fluid (CSF) protein, half had evidence of slight amounts of production of intrathecal antibody to the spirochete, and 70% had one or more of both abnormalities.
 "In addition, more than 50% had abnormal EMGs indicating polyneuropathy affecting both proximal and distal nerve segments, and MRI brain scans showing areas of increased T2 signal intensity. In other words, many of our patients had memory impairments on their psychological assessments, had abnormal CSF anaysis, frequently accompanies by EMG evidence of an axonal neuropathy."

1993: Steere does a 180, publishing THE OVERDIAGNOSIS OF LYME DISEASE.
 Suddenly, that kind of Lyme Allen Steere talked about before (chronic, neurologic, and seronegative), where once he warned were "perilously close to CFIDS and Fibro," now actually were CFIDs and Fibro and therefore a psychiatric illness. In this report, Allen Steere states:
"We thought that 452 (57%) of the 788 patients had another illness rather than Lymedisease. They had had symptoms for a mean duration of 3 years (range, 1 month to 22 years) at the time of our evaluation. More than half of these patients had chronic illnesses with prominent subjective symptoms, such as chronic fatigue syndrome or fibromyalgia. 
"The sex ratio among these patients was 2 to 1 in favor of women, but the age distribution was similar to that in patients with past or current Lyme disease. Although psychiatric disorders such as anxiety, depression, or somatization clearly played a role in the illness of some of these patients, we did not attempt to make psychiatric diagnoses. 
 "Most of the 452 patients had had multiple serological tests for Lyme disease done in multiple laboratories. Of the 452 patients, 203 (45%) had previously had at least on e positive result in another laboratory, often in a borderline-positive range. In our laboratory, none of these patients was seropositive."
Allen Steere has done a complete 180. Where he once acknowledged the multi-system, protean, chronic disease that is similar to lymphoma, he is now calling it a hysterical hypochondriacal women's disease. 

1993: Steere commits research fraud in Europe.
 In the report "Antibody Responses to the Three Genomic Groups of Borrelia burgdorferi in European Lyme Borreliosis," Allen Steere falsified the testing in Europe.
 He used bogus high passage strains, leaving OspA and B out. This means that the bugs have been cultured through many generations and in so doing, they have dropped the plasmid that is encoded with OspA and B. 
 Steere also added the ELISA as a screening test, averaging the concentration signal between the very low Neuro-Lyme responders and the very high autoimmune knee and acrodermatitis responders. As a result, the new, Dearborn, 2-tiered cutoff for an ELISA, will exclude all neurologic cases and of course, most of the IgM responders--which, to Steere, meant persisting infection in 1986.

1993: Barbour & Fish reveal that OspA vaccine trials are underway and start trashing Lyme victims.

 Here we learn that the Phase I and Phase II trials of the OspA vaccines are underway, while Alan Barbour (owner of the ImuLyme OspA patent) and Durland Fish (founding member of the proceed to trash victims of Chronic Lyme/Sepsis, saying it is a disease of hysteria or catastrophizing...because they know by now that OspA is making people sick, especially after 2 injections.  
"However, if a skin lesion is absent, a situation that may occur in 10% or more of infections [4,5], nothing in the clinical presentation can clearly distinguish early Lyme disease from other acute, febrile summer illnesses of temperate latitudes. Later manifestations of Lyme disease, such as arthritis or carditis, can be attributed to other disorders. Neurologic symptoms, especially those involving changes in cognitive functions, are especially difficult to interpret [69- 71]. Moreover, factors such as the premorbid personality and a tendency to somatization may determine the length of convalescence and the response to postinfection fatigue and joint aches [71,72]. Even if the original diagnosis of Lyme disease is undisputed, lingering or recurrent symptoms, many of which are also characteristic of chronic fatigue syndrome or fibromyalgia, may not be attributable to persistent infection."
"Decisions on diagnostic criteria, treatment strategies, research-funding allocation, and insurance reimbursement are being made. Policy-makers are under pressure from some health professionals and lay persons who believe that the spectrum of B. burgdorferi disease is broader than the limits accepted by most peer-reviewed medical journals. The conditions in this larger set include degenerative, inflammatory, and neuropsychiatric conditions not previously thought to be ameliorated by antibiotics [81]. Alternative views of diagnostic criteria and treatment strategies have been presented primarily at regional meetings sponsored by patient advocacy groups and in newsletters devoted to Lyme disease. More recently, the influence of these points of view on the last international scientific meeting on Lyme disease was such that several additional abstracts, which had originally been rejected, were permitted presentation [82]."

1994: FDA vaccine meeting. Dattwyler recommends using serial Western blots. This is apparently ignored.

In this transcript of the 1994 FDA LYMErix meeting, Raymond Dattwyler repeatedly argued against the intended proposal for an arthritic-knee-only case definition for Lyme disease.
Note that this took place in June 1994, which was before Dearborn, so the serodiagnosis business was still up in the air. 

1994: The CDC's "Dearborn" conference, which wasn't about "standardization," but falsification.

 See the invitation here
 What happened was, the labs were invited to "participate in the proceedings!" but then the Lyme criminals blew off all the other labs' recommendations, which, on average, said that the Steere proposal was only 15% accurate--Gary Wormser included.  This shows that the FALSE intent was to have a consensus conference, but these criminals never intended to listen to any other participants' proposals.
In the Dearborn Conference Booklet, you will see who said what, that the "Steere in Europe" report is missing, and that there was no consensus or agreement.  Towards the end of the booklet see what all the contributing labs said about Steere's and the CDC's proposal for a new diagnostic standard. Everyone said it sucked except MarDx who had been given arthritis positive blood to "qualify" their Western Blot test strips, but even they said OspA and B should not be left out of the case definition.

1995: CDC's falsified case definition is officially published.

 1995 "DEARBORN" "Case Definition" published by the CDC in the MMWR (also see here, just in case the link becomes broken), even though all the labs at Dearborn said 'no way.' OspA (band 31) and B (band 34) are left out, as are most IgM bands.
You will see by looking at the October 1994 Dearborn booklet what all the labs said about the proposal for the 2-tiered testing and Steere’s 5 out of 10 band WB criteria, and then as much as we have in detail about the June 1994 FDA meeting on LYMErix, that there was no consensus that Lyme was just a bad knee. In fact, it was a fight and an argument the whole way. 
 Eventually the CDC officers who own patents and who were associated with the fake non-profit ALDF got their way and had their fake vaccines. And later, the bogus Klempner "study" which was based on the Dearborn case definition (the grant for this study was a "fraud on the government"), but the bad guys knew all along that the disease was post-sepsis and was caused by the fake OspA vaccines alone. Certainly Steere knew that late, chronic neurologic Lyme was like post-sepsis since he wrote about it in 1986 and 1990 as you have seen.

1998: FDA approves LymeRIX with numerous provisos.

 LYMErix is declared 76% effective (says Medline). Lyme disease was first called “Lyme Borreliosis” before tick borne disease began to be commercialized after the Bayh-Dole Act came into being, and was only called “Lyme disease” after the Dearborn Conference in 1994.  
LYMErix was approved for use in ages 15-70 and recommended in endemic areas.  At one point they tried to get it approved for use in children; fortunately that never happened.  
The provisos (contraindications) included: Pregnant or breastfeeding mothers, children younger than 15 and adults over 70.
You want to read the entire 1998 FDA meeting transcript. Sikand claims Lyme is hard to diagnose and treat. Schoen says Lyme is underreported by 10-12 times, and Luft is going at these bastards over their disease definitions the entire time, as well as, says the vaccine injuries look exactly like the "protean" or multi-system disease definition and not the "bad knee only" definition. 

1999: Lyme support groups begin receiving phone calls from patients who had “Lyme again” after getting LYMErix.

        Not surprising, after all that we have seen in this timeline.

1999: Gary Wormser reports that OspA is immunosuppressive.

 In his 1999 study (published in 2000) "Modulation of lymphocyte proliferative responses by a canine Lyme disease vaccine of recombinant outer surface protein A (OspA)," he’s talking about how OspA causes immunosuppression in dogs too. The dog vaccines only tolerize against arthritis; they do not prevent spirochetes.
"The effect of the recombinant OspA dog vaccine preparation on human lymphocyte proliferative responses was investigated. Cultures of mitogen- or antigen-stimulated normal human PBL were supplemented with the OspA vaccine preparation at various concentrations. Fig. 1A reveals a dose-dependent regulatory effect of OspA on the proliferation of ConA-stimulated PBL, as compared to controls using saline in place of OspA or unsupplemented PBL cultures. An approximately 25-35% reduction of proliferation was observed with the OspA preparation supplemented at 0.1% volume, while a maximum reduction of 50-60% (P T 0.01) was observed with supplementation at 7% or more (Fig. 1B). 

2001: FDA LYMErix meeting. Whistle blown on Dearborn and OspA.

 Read the transcript of this meeting, where Ben Luft talks about the "twilight zone of disconnect" between what the patients say are vaccine injury, compared to what SKB says. SKB also mentions a high rate of pregnancy failure (miscarriage) at this hearing. SKB (Yves Lobet) also makes fun of Allen Steere and calls him basically an idiot who makes no sense with his theories.
This is also the meeting where Kathleen Dickson blew the whistle on Dearborn and OspA. Her testimony is here, minus the 19 pages that Nancy Cherry "accidentally" omitted.  

2001: Klempner's fake retreatment study in which 2/3 of the participants had not received prior treatment.

 Mark Klempner’s 2001 long-term treatment study (90 days total) was and always will be scientific fraud. 
There were never enough participants; he used invalid methods; he was lying about his previous findings; he was also lying about finding "no DNA-positive patients" among the previously treated because he threw the DNA positive victims out of the study.  Two-thirds of the patients had never been treated before; he never reported what primers he used; he did not look for commonly activated viruses in these patients that are well known to be reactivated in the presence of immune suppression; and no scientifically valid assessments of outcomes of this standard of care "study" were performed. The result was that extended treatments were not beneficial to patients.

2002: LYMErix pulled off the market via FDA ultimatum.

 In the end, the FDA gave SmithKline an ultimatum- either take LYMErix off the market or they would.  To this day they still lie and say that it was due to low sales.  If sales were plummeting, it was due to word getting out that the vaccine was making people sick.
2003: Complaint filed with the USDOJ.
 In 2003, at the advice of staff lawyers for Richard Blumenthal, who at the time was Attorney General for the state of Connecticut, our whistleblower filed a complaintwith the U.S. Department of Justice. Fourteen years later, the charges of scientific fraud and racketeering have gone unanswered, while Lyme disease morbidity and mortality has exploded unchecked by the institutions—namely, the CDC, HHS and state & local health departments— whose duty it is to protect the health of U.S. citizens.

2006: Blumenthal's antitrust investigation into IDSA.

Now Senator Richard Blumenthal sued the Cabal for AntiTrust while he was Connecticut Attorney General.  He did not include the FRAUD part about Dearborn, presumably because he had no "experts" among the "LLMDs" or ILADS to help him with that.  He still doesn't, since ILADS is brainless and do not care what is true.

2015: TRUTHCURES is conceived after our first DC trip to meet with legislators.

 It was on this trip, in June 2015, that then-Senator Jeff Sessions' legal staff told us to convince the U.S. Senate Committee on the Judiciary to hold a hearing for referral of the Lyme Cryme to the Department of Justice. 

2016: Crooks publish (again) that Lyme is an immunosuppression disease.

 Lyme Cabal members Gary Wormser, Allen Steere, “CDC officer” Paul Mead, and others admit Late Lyme and LYMErix diseases are immunosuppression outcomes, saying the “TLR2/1 agonism” (immunosuppression) is probably the “more important” driver of the disease outcome, here.
“This finding suggests that there is redundancy in the ability of the innate immune system to recognize B. burgdorferi and/or that these components can activate pathways that produce anti-inflammatory cytokines……the anti-inflammatory effects might be the more important function of TLR signaling.”
You ready to rise up yet?