Thursday, December 21, 2017

LYME is OspA is Pam3Cys, and is a FUNGAL SHEDDER and THEY KNOW IT











So, Thimerosal was invented to prevent LYMErix in a vial. LYMErix is OspA is Pam3Cys, and is FUNGAL, because it is a TLR2/1 agonist, and the majority of the antigens handled by TLR2/1 are triacyl lipoproteins which mainly come from fungal pathogens or fungal-antigen-shedding spirochetes. I am not making this up. This is how lame the HHS.gov is.
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Why is the growth of fungal antigens together in a vial with live, allegedly attenuated viruses bad?
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Because when you inject live viruses together with immune suppressing fungal antigens (TLR2/TLR1- agonists), you’re not going to get a proper immune response (antibodies) against these live viruses, and then they can revert to “the wild type” – the aggressive disease type -, or even not. They can be virulent (cause damage) as they are, in the allegedly “attenuated” form.
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Who says this?
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None other than Johns Hopkins’ Paul Auwaerter, one of the banes of all the sick and disabled, especially with those with tick bite sepsis, or are among the >/= 50% of people who never recover from “Lyme.”
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Dont believe me?
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Use your browsers search feature to look for his name and the things he has done and published, here:
http://www.actionlyme.org/2017_Criminal_Charges_Sheets_All.…
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But I cant force you to, can I? If you were to do that – search for Auwarter’s journal articles in the charge sheets, it would be of your own will. You’d be an unlazy person willing to go the extra step. You’d be a person who is aware that you cant see the world on a “mobile device,” and you certainly cant blog from one….
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Altered virulence of vaccine strains of measles virus after prolonged replication in human tissue.
Valsamakis A1, Auwaerter PG, Rima BK, Kaneshima H, Griffin DE.
“…Our data suggest that the adverse outcomes associated with immunization of patients suffering from congenital and acquired immunodeficiency syndromes are due to the emergence of an MV strain with increased virulence in a host unable to mount a sufficient immune response to clear the originally inoculated vaccine virus. This situation is mimicked in the SCID-hu mouse. Sequence analyses of pMor-1 H and M and other isolates derived from immunodeficient patients demonstrate that these human tissue-passaged vaccine isolates are highly related to parent vaccine strains (1, 15).
“…However, fatal infections have been documented in immunodeficient children vaccinated with these strains (1, 12, 14, 15). The symptoms of infection occur many months after immunization, and the viruses isolated are similar to the original LA vaccine (1, 15), suggesting that in the absence of an effective host immune response, persistent infection with the vaccine strain can lead to fatal disease. Viruses isolated from these children could potentially represent virulent revertants of the original LA vaccine.”
https://www.ncbi.nlm.nih.gov/pubmed/10482633
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Well, well, well, doesn’t that sound exactly like what they went after Andrew Wakefield for saying?
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There’s more from little Pauly Gisbert Auwaerter (in the charge sheets)…
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Paul Auwaerter Also Says (about how measles causes immunosuppression and that the fatal brain infections can come from the vaccines)…
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“Increased virulence of vaccine strains isolated from immunocompromised infants with fatal infections was not evident.”
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J Infect Dis. 1999 Oct;180(4):950-8.
Measles virus infection in rhesus macaques: altered immune responses and comparison of the virulence of six different virus strains.
Auwaerter PG1, Rota PA, Elkins WR, Adams RJ, DeLozier T, Shi Y, Bellini WJ, Murphy BR, Griffin DE.
“Measles remains a major cause of childhood mortality, with questions about virus virulence and pathogenesis still requiring answers. Rhesus macaques were infected with 5 different culture-adapted strains of measles virus, including 2 from patients with progressive vaccine-induced disease, and a sixth nonculture-adapted strain, Bilthoven. All caused infection detectable by reverse transcriptasepolymerase chain reaction and induction of antibody. Chicago-1 and Bilthoven induced viremias detectable by leukocyte cocultivation. Bilthoven induced Koplik’s spots, conjunctivitis, and rash.
Lymphopenia and depressed interleukin (IL)-2 production were followed by monocytosis and eosinophilia. All monkeys, including 41 involved in a primate facility outbreak, showed suppressed responses to phytohemagglutinin. As the rash resolved production of IL-2, IL-1beta, tumor necrosis factor-alpha, IL-6, and IL-5 mRNA increased. Monkeys are useful for studies of measles immunopathogenesis, but virus strains must be carefully chosen. Increased virulence of vaccine strains isolated from immunocompromised infants with fatal infections was not evident.
”Measles is an important human disease that causes the death of ∼1,000,000 children [a million, really, Paul?- kmd] each year. Most of these deaths are due to secondary infections [1] {{sound familiar?- it should, that is what happens in Lyme or tick-bite-sepsis- kmd}}.
“This increase in susceptibility to other pathogens is associated with a well-documented measles-induced immunosuppression [2]. This suppression of immune responses is incompletely understood and is probably multi-factorial: it
is likely that different mechanisms are of primary importance in early and late phases of infection. Human studies of necessity focus on the time of the appearance of the rash and thereafter, because that is when measles is recognized clinically. Studies in primates offer the opportunity to look at all phases of infection.
”Many of the deaths associated with secondary infection could be prevented by more widespread application of measles immunization. The vaccine against measles is a live attenuated virus with an impressive record of efficacy and safety, although suppression of immune responses is often detectable after immunization [3]. …”
https://www.ncbi.nlm.nih.gov/pubmed/10479117
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So, the vaccine strains, allegedly rendered un-virulent, are, in fact, virulent. Thanks, Paul, we will let everyone know.
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And so, the measles virus, itself, injected into the children with numerous other active viruses at the same time, CAN BE the cause of the immunosuppression and the resultant activation of those other vaccine viruses. Check. Good to know.
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So, inject kids first, then find out of they were immunosuppressed afterwards (or because one of the 3, at least, measles can cause this immunosuppression on its own) – by the results, the brain damage. All these MMR viruses are neurotropic and the models of this “neurodevelopmental brain damage” from these live viruses are borna virus or picornavirus (see that also in the charge sheets, by Stanley Plotkin, another Lyme dick).
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Also, Auwaerter says: “This suppression of immune responses is incompletely understood and is probably multi-factorial: it is likely that different mechanisms are of primary importance in early and late phases of infection. Human studies of necessity focus on the time of the appearance of the rash and thereafter, because that is when measles is recognized clinically.”
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So, inject 3 or more live, allegedly attenuated viruses into infants with immature immune systems, some of which are known to cause immunosuppression, and we know what happens in immunosuppression – reactivated viruses.
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Auwaerter is also suggesting there is the non-spots form of the disease, or a non-inflammatory form of the disease. The Lyme criminals say “you cannot have a disease without inflammation,” or that “there is no disease other than autoimmune,” when we know the opposite is the most damaging outcome: live
viruses and infections without immunity. And go ahead on your own and google “reverts to wild type” or just “wild type.”
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Why do they say this about Lyme – that “you cant have a disease unless it is inflammatory or autoimmune (see this Shapiro quote on this in the charge sheets) ?
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Because as you have just seen, that is how the Autism Vaccines work. And there is more such scientific evidence in the charge sheets. The Occam’s Razor charge sheet and the Common Mechanisms charge sheet are related. They’re basically all one continuous charge, and they contain mainly evidence by the criminals of Lyme and CFIDS and Vaccines.
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How are they going to defend themselves in court regarding saying something 180 degrees from their public statements in the scientific literature, especially over EVERY aspect of this? How can they say they were wrong about biomarkers of disease or their general statements about vaccines or how Lyme caused immunosuppression or that they reported that the Dearborn case definition only detected 15% of the cases, and then said publicly, that all else was NOT-LYME?
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But we dont have time to fuss and we cant dumb this down to a level or a class of people who would not even bother to use a dictionary or Wikipedia, or the personality disordered. (Yes, ticks can bite all kinds of people, not just people with the genetic/NF1-related kind of Autism, or normal people or smart people or lazy people or dumb people or handsome people or ugly people or the severely vain or delusional.)
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We have to reach a target audience somewhere between the lay person and the actual scientists we’re quoting in the charge sheets. We cant talk baby talk to any potential young, untarnished physicians who have heard nothing about the Lyme Crymes before. We need people who are willing to put a little effort into this, to study the charge sheets, communicate with the rest of the bloggers, ask questions, and be involved in putting an end to the CDC’s crimes on behalf of their future victims.
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If that’s you, please help us out. “No army in the world can defeat an idea whose time has come” – Victor Hugo.
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December 12, 2017 in Recruiting Bloggers/Activists.
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